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Subsequent studies demonstrated that apelin signaling is highly context dependent, with differential coupling to Gα_i, Gα_q, and β-arrestin pathways depending on ligand structure, receptor density, and tissue environment (Murali and Aradhyam, 2023)
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The Beta-Arrestin Hypothesis# A key insight driving next-generation drug design is the role of beta-arrestin in GLP-1 receptor signaling
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