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The hormone CCK has been particularly well-studied in this context, an interaction which is primarily a paracrine signal of CCK from EECs to CCK1 Receptors (CCK1R) on vagal afferents (Whited et al., 2006
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These frameworks consider factors such as hormone responses (including insulin, GLP-1, and leptin), individual differences in hunger and fullness control, and brain reward processing
